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In this paper, we initially focused on uncovering the mechanism of action of DPI, a NADPH oxidase inhibitor that we previously found to specifically inhibit MNA NB cell proliferation (56, 57)

Inhibition of lipid synthase activity: AOD 9604 reduces the activities of fatty acid synthase (FAS) and acetyl-CoA carboxylase (ACC), and decreases de novo synthesis of fatty acids

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By silencing NNMT, it enables a natural uptick in NAD levels, leading to enhanced mitochondrial function, lean mass preservation, and fat metabolism without overstimulation

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