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Adaptive Regulation of the Brain's Antioxidant Defences by Neurons and Astrocytes. Free Radical Biology and Medicine 100: 147152

m / z (ESI) 436.2569 (M 2+

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Ischemic models induce rapid oxidative damage by reperfusion-induced ROS, allowing detailed investigations into acute oxidative injury mechanisms, tissue-specific antioxidant responses, and therapeutic efficacy of antioxidants and regenerative interventions such as stem cell-derived EVs (Table 1)

Mechanism of actions Skin-Lightening agents: Hydroquinone i nhibits tyrosinase activity, cellular metabolism, affect both DNA and RNA syntheses and melanin Kojic Acid/Kojic acid tripeptides inhibit the catecholase activity of tyrosinase