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Nevertheless, experimental data suggest that the altered intracellular redox status associated with hyperhomo-cysteinemia-induced oxidant stress is the major molecular mechanism responsible for the atherothrombogenicity of moderately elevated circulating homocysteine

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dos Santos Tenrio MC, Gomes Graciliano N, Andra Moura F, Menezes de Oliveira AC, Fonseca Goulart MO

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