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When the relative bioavailability for F1 was compared to that of pure oral drug, it was found to be 9.77

This therapeutic effect was mechanistically linked to reductions in pro-inflammatory cytokines (TNF-, IL-1), enhancement of antioxidant defenses (e.g., increased superoxide dismutase activity), suppression of apoptosis via caspase-3 inhibition, and modulation of NF-B pathwayscollectively yielding neuroprotection and symptomatic relief ( Curcumin shows therapeutic potential for managing peripheral neuropathic pain through multiple mechanisms (Zhang et al., 2020b)

Interplay between frailty and cardiometabolic disorders: from pathophysiology to clinical implications

[PubMed: 33894838] Davies MJ, Bajaj HS, Broholm C, et al

Plant Physiol 141(4):16531665