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ATF5-mediated mitochondrial unfolded protein response protects against Pb-induced mitochondria damage in SH-SY5Y cell

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This upregulation will in turn up-regulate at the transcriptional level the expression of proinflammatory cytokines (IL-1, IL-6, IL-18, etc.), chemokines, adhesion molecules, Fas ligands33 which will activate the caspase cascades (apoptosis) and the ATP depletion from mitochondria (necrosis) leading to an autosustaining and autopotentiating oxidative loop

It supports skin health, which is a crucial boundary and the first line of defense against pathogens entering the body

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