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This is why it remains a cornerstone compound for researchers exploring complex, multi-factorial conditions

Rather surprisingly, this suggests that the benefits of reduced mTORC1 signaling, and increased autophagy may be mediated primarily by an A-independent mechanism and moreover, previous studies have demonstrated that mTORC1 inhibition leads to the suppression of tauopathy 65,66

These generated 33 identified benefits to the AF (see Table 4-1)

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