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IGF-1 reduces serum GH levels (via somatostatin negative feedback in the pituitary) which in turn suppresses GH actions in the liver, thus enhancing insulin actions in this organ [165]
A component-target-disease network was built in Cytoscape 3.10.2
These hallmarks include sustained proliferative signaling, evasion of growth suppressors, resistance to cell death (apoptosis), induction of ferroptosis (a form of cell death caused by iron accumulation and oxidative stress), angiogenesis (formation of new blood vessels to supply tumors), activation of invasion and metastasis, deregulation of cellular energetics, immune evasion, conferring genome instability and mutation, and tumor-promoting inflammation [135,136,137]
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