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Neuroprotective peptides (Cerebrolysin, Thymosin Beta-4) directly support ganglion cell survival

No pathways associated with inflammation were enriched in this analysis

It has been studied in animals for its potential to accelerate tendon, ligament, muscle, and even gut tissue repair

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Across these models the recurring theme, where effects are seen, is not that GHK-Cu creates tissue from nothing, but that it appears to influence the normal repair sequence recruitment of fibroblasts, matrix synthesis, cross-linking supported by copper-dependent lysyl oxidase, and angiogenesis