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5-Aza-2'-Deoxycytidine and valproic acid in combination with CHIR99021 and A83-01 induce pluripotency genes expression in human adult somatic cells

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These studies underscore the therapeutic potential of both protein-coding and noncoding RNAs as targets in OA ferroptosis intervention

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Abnormal levels of metabolites in conjunction with mutations in cancer-related genes can not only lead to the activation of oncogenes, inactivation of tumor suppressor genes, but also result in aberrant functions, protein stability, localization, interactions, and activity within tumor cells [5]