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One possible scenario is the metabolic cofactor NADPH, which is known to be required not only for lipid synthesis but also for the function of antioxidant reductases, such as glutathione reductase (GSR) and thioredoxin reductase (TXNRD) 86

For this reason, genetic mosaicism is currently the most accepted theory to explain this condition, though it has not yet been genetically confirmed.94 New pathogenic pathways and potential new therapeutic targets The pathogenesis of vitiligo is multifactorial, involving a complex interaction between genetic predisposition and various environmental factors, oxidative stress, and autoimmune responses

The result of both disorders may be insulin resistance, impaired glucose tolerance, and weight gain (5, 6)

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