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As PPA is metabolized through the TCAC, we propose that excess exogenous PPA or related short-chain fatty-acid metabolites interfere with mitochondrial metabolism, thereby causing acquired mitochondrial dysfunction

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In addition, miR-513a-3p sensitizes human A549 lung adenocarcinoma cells to chemotherapy by targeting GSTP1 (Zhang et al., 2012)

It also spares the use of amino acids as energy sources during exercise, making them potentially available for new protein synthesis [4] and decreases the accumulation of lactate

It is not surprising then that most attempts to increase intracellular glutathione (GSH) by glutathione supplementation have been unsuccessful [19, 20]