The existing evidence base comes from preclinical (animal) studies conducted primarily at Washington State University, where the compound was developed and patented for potential use in Alzheimer's disease
Sustained glucagon signaling can slow the conversion of thyroid hormone into its active form: Free T3 drops while TSH reads normal, so a standard thyroid panel misses it. Cold intolerance and brain fog stacked on top of fatigue is the pattern that warrants a Free T3, not just a TSH
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