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Further optimization of the timing, dose, and duration of IGF-1R mAb treatment is warranted in order to maximize benefits while minimizing risks, particularly given recent evidence that transient administration of other drugs can have persistent benefits on age-related outcomes 43

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As mentioned, CXCL10, significantly elevated under IFN- induction, guides CD8 + T cells to lesion sites via CXCR3

M2 microglia promotes the release of anti-inflammatory factors, such as TGF- and IL-10, while M1 microglia plays an opposite role (116)