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Essentially (i.e., providing a common vascular disability point), this might correspond to the previously described syndrome commonly seen with an endothelium-damaging agent overdose, alcohol [25], lithium [24], myocardial infarction [23], and maintained intra-abdominal hypertension, grade III and IV [22], as well as corresponding to the described occlusion syndrome with major vessel occlusion, peripherally [11,12,13,14,15,16,17,18,19] or centrally [20]

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Unlike the classical seven-bladed -propeller observed in other DCAFs, DET1 adopts a unique architecture

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