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Clinical studies have demonstrated its effectiveness in obesity treatment, showing significant reductions in body weight, BMI, waist and hip circumference, and body fat, outperforming placebos and setting new standards for weight loss therapies

These key molecules not only provide novel perspectives for elucidating AD pathogenesis but also offer potential targets for developing new neuroprotective agents (223)

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They: Produce collagen Organize extracellular matrix Restore tensile structure In experimental tendon transection models, BPC-157 has been associated with: Increased fibroblast density Enhanced migration into injury site Accelerated granulation tissue formation Proposed signalling involvement includes: FAK (Focal Adhesion Kinase) activation ERK1/2 signalling Src pathway modulation FAK signalling is particularly important because it regulates: Cell adhesion Cytoskeletal reorganization Migration along collagen scaffolds Tendon strength depends not just on collagen quantity but alignment