In vivo retention of G/D-CuP and release of D-CuP To assess the spatiotemporal retention of G/D-CuP, it was topically applied to full-thickness dorsal wounds in diabetic mice
HPMA copolymers: origins, early developments, present, and future
Brain endothelial cell junctions after cerebral hemorrhage: Changes, mechanisms and therapeutic targets
MIF null mice are entirely resistant to the detrimental effects of OVX, implying that MIF acts as a key downstream mediator of both the detrimental effects of estrogen deficiency and the beneficial effects of estrogen replacement on skin wound healing [57, 175, 182]
Notably, tumor cell lines killed by the ANO6 activator melittin were also driven to cell death through ferroptosis when exposed to erastin and RSL3