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Research in pituitary biology has demonstrated that simultaneous activation of both the ghrelin and GHRH receptor pathways produces a synergistic amplification of GH secretion that exceeds the additive effect of activating either pathway alone

PubMed Central (PMC4425239)

IL-1, produced by inflammasome-activated macrophages and granulosa cells, further potentiates this process by inducing secondary cytokine waves and promoting endothelial activation, creating a self-reinforcing inflammatory circuit within the ovarian microenvironment [24, 28]

The restoration of biomechanical properties demonstrated in animal ligament models suggests potential for improved outcomes compared to rest and splinting alone

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