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The sensitivity of PR3-ANCA was comparable to that of atypical perinuclear ANCA, but its specificity was significantly higher, suggesting that PR3-ANCA could serve as a potential biomarker for extensive UC [16, 33, 34]
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The accumulation of high levels of reactive oxygen species, neurotoxic substances, and inflammatory cytokines forms the pathological mechanism underlying CNSD, which results in dysfunction of the nervous system and the development of therapeutic targets for neurological diseases (Patel, 2016)
