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"Likewise, CGRP transcription can be stimulated by endogenous inflammatory molecules, such as TNF-a, which increases the CGRP promoter activity and actuates MAPK pathway

There is therefore an urgent need to identify the subtype-specific molecular mechanisms underlying TNBC progression and resistance to chemotherapy that may represent therapeutic targets

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Nanomedicines reprogram synovial macrophages by scavenging nitric oxide and silencing ca9 in progressive osteoarthritis